cell adhesion molecule造句

• Prokaryotic expression of neural cell adhesion molecule l1 - ig and its function
  的原核表达及其功能
• Liver cell adhesion molecule , lcam
  肝细胞粘合分子
• Structure and action of neural cell adhesion molecules in nervous system
  神经细胞黏附分子结构及其在神经系统的作用
• Study progress in cell adhesion molecule and the pathogenesy of coronary heart disease
  粘附分子与冠心病发病机制研究进展
• The effects of xueshuanxinmaining on cell adhesion molecules in dyslipidemia patients
  血栓心脉宁对高脂血症患者粘附分子的影响
• Expression and significance of the vascular cell adhesion molecule - 1 in cardiac allografts of rats
  1在大鼠心脏移植慢性排斥模型中的表达
• Study on relationship between soluble vascular cell adhesion molecule - 1 and senile multi - infarct dementia
  1与老年多梗死性痴呆关系的研究
• Variation of soluble vascular cell adhesion molecule - 1 in serum of patients with cerebral infarction
  脑梗死患者血清可溶性血管细胞黏附分子1的变化
• Effects of yiqi huayu recipe on neural cell adhesion molecule in rats with lumbar nerve root compression
  益气化瘀方对腰神经根损伤大鼠神经细胞黏附分子的作用
• Catenin binds to the cytoplasmic tail of a transmembrane cell adhesion molecule - epithelial cadherin ( e - cadherin ) in epithelial cells and forms an adhesion complex [ e - cadherin - p - catenin ( or y - catenin ) - cc - catenin ] ( cadherin - catenin - complex , ccc ) , this complex play an important role in maintaining epithelial cell adhesion . some catenins such as - , - and p120ctn catenins are also crucial in cell signaling , they play an important role in cell proliferation and cell apoptosis
  在上皮细胞内，连环蛋白均能与跨膜细胞粘附分子?上皮钙粘蛋白( e - cadherin )的胞内肽段结合而形成了由上皮钙粘蛋白- -连环蛋白( -连环蛋白) - -连环蛋白组成的粘附连接体( adherensjunctions , ajs ) ，这种复合体对维持细胞正常的粘附功能是必需的。
• Conclusion one mechanism of wmt on aso may be its inhibition on arteriosclerosis by way of downregulating the expression of vascular endothelial cells adhesion molecules to decrease the adhesion of monocyte to vec , therefore to inhibit the monocytes migrating into vascular intima to develop foam cells
  结论下调血管内皮细胞黏附分子的表达，减少单核细胞与内皮细胞的黏附，从而抑制单核细胞迁移至血管内膜形成泡沫细胞，抑制动脉粥样硬化as的形成，可能是温脉通治疗aso的机制之一。
• Conclusion one mechanism of wmt on aso may be its inhibition on arteriosclerosis by way of downregulating the expression of vascular endothelial cells adhesion molecules to decrease the adhesion of monocyte to vec , therefore to inhibit the monocytes migrating into vascular intima to develop foam cells . 2007 vol
  结论下调血管内皮细胞黏附分子的表达，减少单核细胞与内皮细胞的黏附，从而抑制单核细胞迁移至血管内膜形成泡沫细胞，抑制动脉粥样硬化as的形成，可能是温脉通治疗aso的机制之一。